What symptom is NOT typically associated with vertebrobasilar infarction?

Vertebrobasilar infarction occurs due to reduced blood flow or occlusion in the vertebral and basilar arteries, which supply the posterior circulation of the brain. This area is responsible for many vital functions, including regulating balance, coordination, and eye movements, which explains why symptoms often involve neurological deficits.

Dizziness, diplopia (double vision), and cranial nerve palsies are commonly observed in vertebrobasilar infarctions. Dizziness results from disruption of the vestibular pathways, while diplopia can occur due to involvement of cranial nerves that control eye movements (such as CN III, IV, or VI). Cranial nerve palsies can arise from the ischemia affecting the brainstem where these nerves reside.

In contrast, loss of sensation in the legs is primarily associated with conditions affecting the anterior circulation (such as strokes involving the middle cerebral artery) or spinal cord conditions. The posterior circulation does not typically cause lower extremity sensory deficits but rather upper extremity symptoms or coordination issues depending on the specific areas involved. Thus, loss of sensation in the legs is not a characteristic symptom of vertebrobasilar infarction, making it the correct choice for this question.